2015年2月28日 星期六

乳腺炎化膿被確診時 ---- 會破皮膚或擠的出來??


全職媽,寶貝1m16d,全母乳

目前深受乳線炎之苦,硬塊佔據一半乳房也化膿了,


現在吃抗生素治療中可是怎麼擠膿都不出來,


真的不想住院打抗生素,想在剩的六天在家把膿跟硬塊處理


好,請有經驗媽媽分享,已經換三家醫


院了......



給媽媽的建議: 1.乳汁移出效果不彰時  要考慮是否乳腺炎處於急性期   結締組織受發炎病


灶波及   此時用藥不夠或種類未針對菌叢時    會拖延病情    更難治療    有可能皮膚破損膿奶未


必從乳頭排出   2. 每位媽媽乳腺炎症狀都不同   感染到的菌也需培養才知道是哪一群   如果不


是目前口服抗生素可以對抗的    還是需要換藥物種類     不是自己能夠用一般程序消除硬塊



以下文獻請媽媽用翻譯軟體看看 



 參考文獻 BREASTFEEDING MEDICINE Volume 9, Number 5, 2014 ª Mary Ann Liebert, Inc. DOI: 10.1089/bfm.2014.9984






乳腺炎診斷定義 : Definition and Diagnosis 

The usual clinical definition of mastitis is a tender, hot, swollen, wedge-shaped area of breast associated with temperature of 38.5C (101.3F) or greater, chills, flu-like aching, and systemic illness.5 However, mastitis literally means, and is defined herein, as an inflammation of the breast; this inflammation may or may not involve a bacterial infection.6,7 Redness, pain, and heat may all be present when an area of the breast is engorged or ‘‘blocked’’/‘‘plugged,’’ but an infection is not necessarily present. There appears to be a continuum from engorgement to noninfective mastitis to infective mastitis to breast abscess.7 (II-2)


誘發因素 Predisposing Factors

 The following factors may predispose a lactating woman to the development of mastitis.7,8 Other than the fact that these are factors that result in milk stasis, the evidence for these associations is generally inconclusive (II-2):
1.  Damaged nipple, especially if colonized with Staphylococcus aureus
2.Infrequent feedings or scheduled frequency or duration of feedings
3.  Missed feedings
4.  Poor attachment or weak or uncoordinated suckling leading to inefficient removal of milk
5.  Illness in mother or baby
 6.Oversupply of milk
 7.Rapid weaning
8.  Pressure on the breast (e.g., tight bra, car seatbelt)
9.  White spot on the nipple or a blocked nipple pore or duct: milk blister or ‘‘bleb’’ (a localized inflammatory response)9
 10.Maternal stress and fatigue

Management 處理

Effective milk removal Because milk stasis is often the initiating factor in mastitis, the most important management step is frequent and effective milk removal: Mothers should be encouraged to breastfeed more frequently, starting on the affected breast. If pain interferes with the let-down, feeding may begin on the unaffected breast, switching to the affected breast as soon as let-down is achieved. Positioning the infant at the breast with the chin or nose pointing to the blockage will help drain the affected area. Massaging the breast during the feed with an edible oil or nontoxic lubricant on the fingers may also be helpful to facilitate milk removal. Massage, by the mother or a helper, should be directed from the blocked area moving toward the nipple. After the feeding, expressing milk by hand or pump may augment milk drainage and hasten resolution of the problem.11 (III) An alternate approach for a swollen breast is fluid mobilization, which aims to promote fluid drainage toward the axillary lymph nodes.12 The mother reclines, and gentle hand motions start stroking the skin surface from the areola to the axilla.12 (III) There is no evidence of risk to the healthy, term infant of continuing breastfeeding from a mother with mastitis.7 Women who are unable to continue breastfeeding should express the milk from breast by hand or pump, as sudden cessation of breastfeeding leads to a greater risk of abscess development than continuing to feed.11 (III) Supportive measures Rest, adequate fluids, and nutrition are important measures. Practical help at home may be necessary for the mother to obtain adequate rest. Application of heat—for example, a shower or a hot pack—to the breast just prior to feeding may help with the let-down and milk flow. After a feeding or after milk is expressed from the breasts, cold packs can be applied to the breast in order to reduce pain and edema. Although most women with mastitis can be managed as outpatients, hospital admission should be considered for women who are ill, require intravenous antibiotics, and/or do not have supportive care at home. Rooming-in of the infant with the mother is mandatory so that breastfeeding can continue. In some hospitals, rooming-in may require hospital admission of the infant.

Pharmacologic management 藥物處置

 Although lactating women are often reluctant to take medications, women with mastitis should be encouraged to take appropriate medications as indicated. Analgesia. Analgesia may help with the let-down reflex and should be encouraged. An anti-inflammatory agent such as ibuprofen may be more effective in reducing the inflammatory symptoms than a simple analgesic like paracetamol/ acetaminophen. Ibuprofen is not detected in breastmilk following doses up to 1.6 g/day and is regarded as compatible with breastfeeding.13 (III) Antibiotics. If symptoms of mastitis are mild and have been present for less than 24 hours, conservative management (effective milk removal and supportive measures) may be sufficient. If symptoms are not improving within 12–24 hours or if the woman is acutely ill, antibiotics should be started.7 Worldwide, the most common pathogen in infective mastitis is penicillin-resistant S. aureus. 14,15 Less commonly, the organism is a Streptococcus or Escherichia coli. 11 The preferred antibiotics are usually penicillinase-resistant penicillins,5 such as dicloxacillin or flucloxacillin 500 mg by mouth four times per day,16 or as recommended by local antibiotic sensitivities. (III) First-generation cephalosporins are also generally acceptable as first-line treatment, but may be less preferred because of their broader spectrum of coverage. (III) Cephalexin is usually safe in women with suspected penicillin allergy, but clindamycin is suggested for cases of severe penicillin hypersensitivity.16 (III) Dicloxacillin appears to have a lower rate of adverse hepatic events than flucloxacillin.17 Many authorities recommend a 10–14-day course of antibiotics18,19; however this recommendation has not been subjected to controlled trials. (III) S. aureus resistant to penicillinase-resistant penicillins (methicillin-resistant S. aureus [MRSA], also referred to as oxacillin-resistant S. aureus) has been increasingly isolated in cases of mastitis and breast abscesses.20–22 (II-2) Clinicians should be aware of the likelihood of this occurring in their community and should order a breastmilk culture and assay of antibiotic sensitivities when mastitis is not improving 48 hours after starting first-line treatment. Local resistance patterns for MRSA should be considered when choosing an antibiotic for such unresponsive cases while culture results are pending. MRSA may be a communityacquired organism and has been reported to be a frequent pathogen in cases of breast abscess in some communities, particularly in the United States and Taiwan.21,23,24 (I, II-2) At this time, MRSA occurrence is low in other countries, such as the United Kingdom.25 (I) Most strains of methicillinresistant staphylococci are susceptible to vancomycin or trimethoprim/sulfamethoxazole but may not be susceptible to rifampin.26 Of note is that MRSA should be presumed to be resistant to treatment with macrolides and quinolones, regardless of susceptibility testing results.27 (III) As with other uses of antibiotics, repeated courses place women at increased risk for breast and vaginal Candida infections.

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